Neurodegenerative disease is the fastest-growing cause of death in the world. It will soon be the leading cause of death because of cover-ups and corruption at the highest levels.
Deadly proteins are at the heart of the epidemic, which is technically known as transmissible spongiform encephalopathy (TSE). The operative word is “transmissible.” Infectious proteins known as prions (PREE-on) are driving the epidemic. Prions are unstoppable, especially those shed from humans. Prions migrate, mutate and multiply and they become more aggressive and more deadly long the way. TSE is striking down people of all ages. Rogue proteins also appear to play a role in the surging autism epidemic.
Dr. Stanley Prusiner, an American neuroscientist from the University of California at San Francisco, earned a Nobel Prize in 1997 for discovering and characterizing prions and prion disease. President Obama awarded Prusiner the National Medal of Science in 2010 to recognize the importance of his research. Important reforms to policies to protect public health, however, have been elusive.
Prusiner claims that all forms of TSE are caused by infectious prions.
“Alzheimer’s disease (AD) is the most common neurodegenerative disease in humans and will pose a considerable challenge to healthcare systems in the coming years,” Prusiner said. Aggregation of the β-amyloid (Aβ) peptide within the brain is thought to be an initiating event in AD pathogenesis. Many recent studies in transgenic mice have provided evidence that Aβ aggregates become self-propagating (infectious) during disease, leading to a cascade of protein aggregation in the brain, which may underlie the progressive nature of AD. The ability to self-propagate and the existence of distinct strains reveals that Aβ aggregates exhibit many properties indistinguishable from those of prions composed of PrPSc proteins. The evidence that Aβ can become a prion during disease, Aβ prions may be important for understanding the pathobiology of AD.”
Like prion diseases, misfolded alpha-synuclein and tau protein associated with Parkinson’s disease and Alzheimer’s disease, respectively, have been reported in skin tissues of patients with these conditions. Skin could serve as a screen for early diagnosis, but also for monitoring the accumulation of the misfolded proteins in the brain of these neurodegenerative diseases. It also could be another pathway of transmission.
“I learned that scrapie, Creutzfeldt-Jakob disease and kuru had all been shown to be transmissible by injecting extracts of diseased brains into the brains of healthy animals,” Prusiner said. “The infections were thought to be caused by a slow-acting virus, yet no one had managed to isolate the culprit. Whether changes in protein shape are responsible for common neurodegenerative diseases, such as Alzheimer’s, remains unknown, but it is a possibility that should not be ignored.”
“There has been a resurgence of this sort of thinking, because there is now real evidence of the potential transmissibility of Alzheimer’s disease,” says Thomas Wiesniewski M.D. a prion and Alzheimer’s disease researcher at New York University School of Medicine. “In fact, this ability to transmit an abnormal conformation is probably a universal property of amyloid-forming proteins (prions).”
Meanwhile, government and industry are playing dumb in light of growing evidence about reckless policies and practices that are contributing to the epidemic. As such, misinformation and mismanagement are fanning the flames of a global public health disaster that’s spreading like wildfire. The human epidemic is so bad, it’s even killing wildlife. Sick animals are serving as the proverbial canary in a coal mine, while millions of people are being exposed and killed each year. Each victim spreads the disease.
Meanwhile, the medical world is protecting some people from infectious prion disease, but they aren’t protecting everyone from these neurotoxins. For example, neurologists rarely have physical contact with their patients. They are making diagnoses from across the room based on the presenting symptoms. If they observe a movement disorder, the diagnosis is Parkinson’s disease. If they see a memory disorder, it’s Alzheimer’s disease. If the person is totally incapacitated, the diagnosis is Creutzfeldt-Jakob disease. It’s about as scientific as pinning the tail on the donkey with a blindfold on.
Regardless of the diagnosis, these patients are all being sent home to die. Family and caregivers are not warned about the risks of caring for someone with neurodegenerative disease. It’s criminal negligence at best. Even patient advocacy organizations, including the CJD Foundation, are failing to sound the alarm. They are merely trying to calm fears about the disease as opposed to prevent it. Read what the science says and then compare it to the deliberate lies issued by gatekeepers.
Read the full story about CJD and transmissibility.
Gary Chandler is the CEO of Crossbow Communications. He also is the founder of Sacred Seedlings and Earth News He is the author of 11 books about health and environmental issues from around the world. Chandler also is connecting the dots to the global surge in neurological disorders.